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Treatment of AR-positive LNCaP prostate cancer cells with the synthetic steroid R1881 induced transcriptional upregulation of glucose transporters (GLUT1, GLUT12) and glycolytic enzymes (HK1/2, and PFKB2), increased glucose uptake, glucose entry into glycolysis, and glucose storage into lipids ( de novo lipogenesis ) ( Enhanced aerobic glycolysis in response to androgen withdrawal is also observed in in vivo models

There are two possibilities