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The heterozygous genotype ranged from 30% to 50% among whites, 24% to 44% among Asians, and 46% among African-Americans

To mitigate toxicity from mutant SF3B1 expression, an inducible Cre-recombinase system was employed, allowing edited cells to expand before activating mutant allele expression

The glutathione is involved transiently in the reactions they catalyze

Genetic Predisposition: Some people may have a genetically higher risk of developing this disease