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His genetic test revealed his GIPR variant responds well to GLP-1 therapy, suggesting he's a strong responder

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Its therapeutic value lies in its ability to selectively induce apoptosis in senescent cells by disrupting the FOXO4-p53 interactome, a mechanism that evades the collateral cytotoxicity often seen with broader senolytics like Navitoclax (Huang et al., 2021)

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