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If anemia and symptoms are severe, early labs (e.g

Abnormalities in the structural components can disrupt the skin barrier and destabilize skin homeostasis, causing various cutaneous diseases (Yosipovitch et al., 2019)

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To address this question, we mutated Tyr110 in the CPYS-motif of ScGrx7 as a candidate residue that might contribute to the glutathione activator site (see Discussion for details) and performed a preliminary study with wild-type ScGrx7 as a control

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