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Mechanistically, the NF-kB and cytokine effects should theoretically help, but lupus pathogenesis involves pathways (particularly type I interferon and complement activation) that may not be as responsive to GLP-1 signaling

Collectively, these mechanisms target critical nodes implicated in HSV-mediated immunopathology, suggesting a potential role in mitigating lesion-associated tissue damage and inflammatory pain

However, these uncontrolled observations require validation through prospective, controlled clinical trials before establishing standard treatment durations for specific clinical indications

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doi: 10.1016/j.isci.2022.104630 57 LiXCaiPTangXWuYZhangYRongX