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Interpretable deep learning translation of GWAS and multi-omics findings to identify pathobiology and drug repurposing in Alzheimers disease

There is considerable interest in pairing GLP-1R agonism with glucagon receptor agonism to enhance both energy expenditure to maximize weight loss and potentially leverage the lipid oxidative properties of glucagon in hepatocytes to target nonalcoholic fatty liver disease

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The FDA has documented hundreds of adverse event reports from compounded GLP-1 dosing errors, and compounded products come with provider oversight that research peptides entirely lack

However, many of these claims are based primarily on anecdotal reports and preclinical research rather than large human clinical trials