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In parallel, therapeutic strategies should focus on targeting the shared mechanisms of neurotoxicity, such as oxidative stress and neuroinflammation, while considering drug-specific interventions

To test whether the regulation of CYP8B1 by T3 would have any effect on BA composition, we performed UFLC-Triple-time of flight/MS-based metabolite profiling to quantitate the BA levels and found that the percentage of 12-hydroxylated (12-OH) BAs was reduced, while the proportion of non-12-hydroxylated (non-12-OH) BAs was increased in the gallbladder bile, ileum, liver, serum, and urine after five days of T3 treatment in MMI mice (Fig

5 Such trials will be essential to clarify the primary immunologic effects of GLP-1 RAs, offering clearer insight into their therapeutic potential beyond glycemic control

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