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We monitored ROS production, modifications in mitochondrial area and respiratory parameters from macrophages following sUA stimulus and observed that sUA is released in a hypoxic environment and, when the first signal of inflammasome activation is present, is able to trigger NLRP3 through the production of mitochondrial ROS

BPC-157 restores balanced NO activity by stabilizing endothelial nitric oxide synthase (eNOS) while suppressing overactivation of inducible nitric oxide synthase (iNOS)reducing oxidative stress and improving microcirculation

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