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The ICMR-INDIAB study places it at 11.4%, while NFHS-5 estimates it at 16.1%

Improvements tend to be modest and inconsistent, depending on baseline fertility, dose, and treatment duration

identified the previously unrecognized SLC25A45 as a mediator of TML transport into mitochondria, thereby enabling de novo l -carnitine biosynthesis

Self-reported community sources describe GI events scaling with per-dose amount and brief fatigue scaling with total daily dose

Moreover, MnSOD is thought by virtue of its localization to provide the principal source of protection against oxidative damage provoked by mitochondrial respiration