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Acknowledgements This work was supported by the Austrian Science Fund (10.55776/COE16, T.H.), the Swedish Research Council (2023-03058, T.H.), the Swedish Brain Foundation (FO2022-0300, T.H.), the Novo Nordisk Foundation (NNF23OC0084476, T.H.) and the European Research Council (ERC-2020-AdG-101021016

Research Applications of the BPC-157 Peptide Gastric and intestinal mucosal injury models, where BPC-157 is the standard reference compound for studying cytoprotection independent of acid-suppression mechanisms

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Second, as mentioned above, c-Met inhibitors which target the interaction between HGF and c-Met may lose efficacy owing to cell attachment (Wang et al., 2001), gene amplification of c-Met, DCP binding to c-Met (Suzuki et al., 2005), gene mutation in the c-Met activation loop (Steinway et al., 2015)

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