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Therefore, this study aims to investigate the interplay between epigenetic modifications, oxidative stress, and trace element imbalances in the pathogenesis of polycystic ovary syndrome using a dehydroepiandrosterone (DHEA)-induced rat model

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Identity verification of each peptide (BPC157, TB500, GHKCu) by mass spectrometry and sequence analysis prior to blending

147 1.71 and 171 2.57 at doses of 25, 50 and 100 mg/kg

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